UNAUTHORED PUBLICATIONS RELATED TO RESEARCH

INTERESTING PUBLICATIONS (UNAUTHORED)

PBZ Formatted

Ahlqvist K.J., A. Suomalainen, and R.H. Hämäläinen. 2015. Stem cells, mitochondria and aging. Biochim Biophys Acta - Bioenerg 1847:1380–1386.
Ashcroft G.S., S.J. Mills, and J.J. Ashworth. 2002. Ageing and wound healing. Biogerontology 3:337–345.
Austad S.N. 2010. Methusaleh’s Zoo: How Nature provides us with Clues for Extending Human Health Span. J Comp Pathol 142:S10–S21.
Austad S.N. and J.M. Hoffman. 2018. Is antagonistic pleiotropy ubiquitous in aging biology? Evol Med Public Heal 2018:287–294.
Baar M.P., E. Perdiguero, P. Munoz-Canoves, and P.L.J. De Keizer. 2018. Musculoskeletal senescence : a moving target ready to be eliminated 147–155.
Bacman S.R., J.H.K. Kauppila, C. V. Pereira, N. Nissanka, M. Miranda, M. Pinto, S.L. Williams, et al. 2018. MitoTALEN reduces mutant mtDNA load and restores tRNAAla levels in a mouse model of heteroplasmic mtDNA mutation. Nat Med 24:1696–1700.
Bertolotti M., A. Lonardo, C. Mussi, E. Baldelli, E. Pellegrini, S. Ballestri, D. Romagnoli, et al. 2014. Nonalcoholic fatty liver disease and aging: Epidemiology to management. World J Gastroenterol 20:14185–14204.
Brunk U.T. and A. Terman. 2002. The mitochondrial-lysosomal axis theory of aging: Accumulation of damaged mitochondria as a result of imperfect autophagocytosis. Eur J Biochem.
Campbell R.D., F. Rosell, C. Newman, and D.W. Macdonald. 2017. Age-related changes in somatic condition and reproduction in the Eurasian beaver: Resource history influences onset of reproductive senescence. PLoS One 12:1–23.
Chinopoulos C., S.F. Zhang, B. Thomas, V. Ten, and A.A. Starkov. 2011. Isolation and Functional Assessment of Mitochondria from Small Amounts of Mouse Brain Tissue. Methods Mol Biol 793:311=324.
Clark K.A., D.K. Howe, K. Gafner, D. Kusuma, S. Ping, S. Estes, and D.R. Denver. 2012. Selfish little circles: Transmission bias and evolution of large deletion-bearing mitochondrial DNA in caenorhabditis briggsae nematodes. PLoS One 7:1–8.
Derting T.L. 1996. Changes in gastrointestinal characteristics of an omnivorous species, the raccoon, with lactation and season. J Mammal 77:440–448.
Drenos F. and T.B.L. Kirkwood. 2005. Modelling the disposable soma theory of ageing. Mech Ageing Dev 126:99–103.
Franceschi C., P. Garagnani, C. Morsiani, M. Conte, A. Santoro, A. Grignolio, D. Monti, et al. 2018. The continuum of aging and age-related diseases: Common mechanisms but different rates. Front Med 5.
Franceschi C., P. Garagnani, G. Vitale, M. Capri, and S. Salvioli. 2017. Inflammaging and ‘Garb-aging.’ Trends Endocrinol Metab 28:199–212.
García‐Ruiz C. and J.C. Fernández‐Checa. 2018. Mitochondrial Oxidative Stress and Antioxidants Balance in Fatty Liver Disease. Hepatol Commun 2:1425–1439.
Gavrilov L.A. and N.S. Gavrilova. 2002. Evolutionary Theories of Aging and Longevity. Rev Artic TheScientificWorldJOURNAL 2:339–356.
Gomes A.P., N.L. Price, A.J.Y. Ling, J.J. Moslehi, M.K. Montgomery, L. Rajman, J.P. White, et al. 2013. Declining NAD+ induces a pseudohypoxic state disrupting nuclear-mitochondrial communication during aging. Cell 155:1624–1638.
Harman D. 1992. Free radical theory of aging. Mutat Res DNAging.
Hayflick L. 1984. Intracellular determinants of cell aging. Mech Ageing Dev 28:177–185.
Herai R.H., P.D. Negraes, and A.R. Muotri. 2017. Evidence of nuclei-encoded spliceosome mediating splicing of mitochondrial RNA. Hum Mol Genet 26:2472–2479.
Hiruki L.M., I. Stirling, W.G. Gilmartin, T.C. Johanos, and B.L. Becker. 1993. Significance of wounding to female reproductive success in Hawaiian monk seals (Monachus schauinslandi) at Laysan Island. Can J Zool 71:469–474.
Hood W.R., A.S. Williams, and G.E. Hill. 2019. An Ecologist’s Guide to Mitochondrial DNA Mutations and Senescence. Integr Comp Biol 59:970–982.
Hood W.R., Y. Zhang, A. V. Mowry, H.W. Hyatt, and A.N. Kavazis. 2018. Life history trade-offs within the context of mitochondrial hormesis. Integr Comp Biol 58:567–577.
Irvine K.M., R. Skoien, N.J. Bokil, M. Melino, G.P. Thomas, D. Loo, B. Gabrielli, et al. 2014. Senescent human hepatocytes express a unique secretory phenotype and promote macrophage migration. World J Gastroenterol 20:17851–17862.
Itsara L.S., S.R. Kennedy, E.J. Fox, S. Yu, J.J. Hewitt, M. Sanchez-Contreras, F. Cardozo-Pelaez, et al. 2014. Oxidative Stress Is Not a Major Contributor to Somatic Mitochondrial DNA Mutations. PLoS Genet 10.
Kakimoto Y., C. Okada, N. Kawabe, A. Sasaki, H. Tsukamoto, R. Nagao, and M. Osawa. 2019. Myocardial lipofuscin accumulation in ageing and sudden cardiac death. Sci Rep.
Kauppila J.H.K., N.A. Bonekamp, A. Mourier, M.A. Isokallio, A. Just, T.E.S. Kauppila, and J.B. Stewart. 2018a. Base-excision repair deficiency alone or combined with increased oxidative stress does not increase mtDNA point mutations in mice 46:6642–6669.
Kauppila J.H.K. and J.B. Stewart. 2015. Mitochondrial DNA: Radically free of free-radical driven mutations. Biochim Biophys Acta - Bioenerg 1847:1354–1361.
Kauppila T.E.S., A. Bratic, M.B. Jensen, F. Baggio, L. Partridge, H. Jasper, S. Grönke, et al. 2018b. Mutations of mitochondrial DNA are not major contributors to aging of fruit flies. Proc Natl Acad Sci U S A 115:E9620–E9629.
Kennedy G.C. 1958. Liver Growth in the Lactating Rat. J Endocrinol 17:158–60.
Kirkland J.L., T. Tchkonia, Y. Zhu, L.J. Niedernhofer, and P.D. Robbins. 2017. The Clinical Potential of Senolytic Drugs. J Am Geriatr Soc 65:2297–2301.
Kojima H., T. Inoue, H. Kunimoto, and K. Nakajima. 2013. IL-6-STAT3 signaling and premature senescence. Jak-Stat 2:e25763.
Krumschnabel G., A. Eigentler, M. Fasching, and E. Gnaiger. 2014. Use of safranin for the assessment of mitochondrial membrane potential by high-resolution respirometry and fluorometry. Methods Enzymol (1st ed., Vol. 542). Elsevier Inc.
Kujoth G.C., P.C. Bradshaw, S. Haroon, and T.A. Prolla. 2007. The role of mitochondrial DNA mutations in mammalian aging. PLoS Genet 3:0161–0173.
Kujoth G.C., C. Leeuwenburgh, and T.A. Prolla. 2006. Mitochondrial DNA mutations and apoptosis in mammalian aging. Cancer Res 66:7386–7389.
Lahdenperä M., K.U. Mar, and V. Lummaa. 2014. Reproductive cessation and post-reproductive lifespan in Asian elephants and pre-industrial humans. Front Zool 11:1–14.
Lakshmanan L.N., Z. Yee, L.F. Ng, R. Gunawan, B. Halliwell, and J. Gruber. 2018. Clonal expansion of mitochondrial DNA deletions is a private mechanism of aging in long-lived animals. Aging Cell 17:1–30.
Martin, William F., Grarg, Sriram, Zimorski V. 2015. Endosymbiotic theories for eukaryote origin. Philos Trans B R Soc 370.
Massudi H., R. Grant, G.J. Guillemin, and N. Braidy. 2012. NAD + metabolism and oxidative stress: The golden nucleotide on a crown of thorns. Redox Rep 17:28–46.
Mikesic D.G. and L.C. Drickamer. 1992. The University of Notre Dame Factors Affecting Home-Range Size in House Mice ( Mus musculus domesticus ) Living in Outdoor Enclosures. Am Midl Nat 127:31–40.
Miller R.A., J.M. Harper, R.C. Dysko, S.J. Durkee, and S.N. Austad. 2002. Longer Life Spans and Delayed Maturation in Wild-Derived Mice. Exp Biol Med 227:500–508.
Montecino-rodriguez E., B. Berent-maoz, K. Dorshkind, E. Montecino-rodriguez, B. Berent-maoz, and K. Dorshkind. 2013. Causes , consequences , and reversal of immune system aging Find the latest version : Review series Causes , consequences , and reversal of immune system aging. J Clin Invest 123:958–965.
Mowry A. V., Z.S. Donoviel, A.N. Kavazis, and W.R. Hood. 2017. Mitochondrial function and bioenergetic trade-offs during lactation in the house mouse (Mus musculus). Ecol Evol 7:2994–3005.
Nissanka N., M. Minczuk, and C.T. Moraes. 2019. Mechanisms of Mitochondrial DNA Deletion Formation. Trends Genet 35:235–244.
Park N.R., H.A. Taylor, V.A. Andreasen, A.S. Williams, K. Niitepõld, K.N. Yap, A.N. Kavazis, et al. 2020. Mitochondrial physiology varies with parity and body mass in the laboratory mouse (Mus musculus). J Comp Physiol B Biochem Syst Environ Physiol 190:465–477.
Pasquali A. 1979. Aggressive responses of isolated mice towards ‘opponents’ of differing social status. Bolletino di Zool 46:41–50.
Poljsak B. and I. Milisav. 2016. NAD+ as the Link between Oxidative Stress, Inflammation, Caloric Restriction, Exercise, DNA Repair, Longevity, and Health Span. Rejuvenation Res 19:406–413.
Prolla T.A. and J.M. Denu. 2014. NAD+ deficiency in age-related mitochondrial dysfunction. Cell Metab. Cell Press.
Ristow M. and K. Schmeisser. 2014. Mitohormesis: Promoting health and lifespan by increased levels of reactive oxygen species (ROS). Dose-Response 12:288–341.
Rolt A., A. Nair, and L.S. Cox. 2019. Optimisation of a screening platform for determining IL-6 inflammatory signalling in the senescence-associated secretory phenotype (SASP). Biogerontology 20:359–371.
Rosshart S.P., B.G. Vassallo, D. Angeletti, D.S. Hutchinson, A.P. Morgan, K. Takeda, H.D. Hickman, et al. 2017. Wild Mouse Gut Microbiota Promotes Host Fitness and Improves Disease Resistance. Cell 171:1015-1028.e13.
Samuels D.C., C. Li, B. Li, Z. Song, E. Torstenson, H. Boyd Clay, A. Rokas, et al. 2013. Recurrent Tissue-Specific mtDNA Mutations Are Common in Humans. PLoS Genet 9.
Sanada F., Y. Taniyama, J. Muratsu, R. Otsu, H. Shimizu, H. Rakugi, and R. Morishita. 2018. Source of Chronic Inflammation in Aging. Front Cardiovasc Med 5:1–5.
Schwartsburd P.M. 1995. On the origin of heterogeneity of lipofuscin fluorophores and their possible interrelations. Gerontology.
Schwartz C.C., K.A. Keating, H. V. Reynolds, V.G. Barnes, R.A. Sellers, J.E. Swenson, S.D. Miller, et al. 2003. Reproductive maturation and senescence in the female brown bear. Ursus 14:109–119.
Schwarzburd P.M. and K.B. Aslanidi. 1991. Resistance of single tumor cells and their intracellular compartments to lipid peroxidation. Med Oncol Tumor Pharmacother 8:57–61.
Selman C., J.D. Blount, D.H. Nussey, and J.R. Speakman. 2012. Oxidative damage, ageing, and life-history evolution: Where now? Trends Ecol Evol 27:570–577.
Serrano M., A.W. Lin, M.E. McCurrach, D. Beach, and S.W. Lowe. 1997. Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16(INK4a). Cell.
Shukla S.P., C. Plata, M. Reichelt, S. Steiger, D.G. Heckel, M. Kaltenpoth, A. Vilcinskas, et al. 2018. Microbiome-assisted carrion preservation aids larval development in a burying beetle. Proc Natl Acad Sci U S A 115:11274–11279.
Speakman J.R. 2008. The physiological costs of reproduction in small mammals. Philos Trans R Soc B Biol Sci 363:375–398.
Stein L.R. and S.I. Imai. 2012. The dynamic regulation of NAD metabolism in mitochondria. Trends Endocrinol Metab 23:420–428.
Stern M. 2017. Evidence that a mitochondrial death spiral underlies antagonistic pleiotropy. Aging Cell 16:435–443.
Szczepanowska K. and A. Trifunovic. 2017. Origins of mtDNA mutations in ageing. Essays Biochem 61:325–337.
Tam Z.Y., J. Gruber, B. Halliwell, and R. Gunawan. 2015. Context-Dependent Role of Mitochondrial Fusion-Fission in Clonal Expansion of mtDNA Mutations. PLoS Comput Biol 11:1–22.
Tapia P.C. 2006. Sublethal mitochondrial stress with an attendant stoichiometric augmentation of reactive oxygen species may precipitate many of the beneficial alterations in cellular physiology produced by caloric restriction, intermittent fasting, exercise and dietary p. Med Hypotheses 66:832–843.
Terman A. and U.T.. Brunk. 2009. Lipofuscin: Mechanisms of formation and increase with age. APMIS.
van Horssen J., P. van Schaik, and M. Witte. 2019. Inflammation and mitochondrial dysfunction: A vicious circle in neurodegenerative disorders? Neurosci Lett 710:6–11.
Vasileiou P., K. Evangelou, K. Vlasis, G. Fildisis, M. Panayiotidis, E. Chronopoulos, P.-G. Passias, et al. 2019. Mitochondrial Homeostasis and Cellular Senescence. Cells 8:686.
Vermulst M., J.H. Bielas, G.C. Kujoth, W.C. Ladiges, P.S. Rabinovitch, T.A. Prolla, and L.A. Loeb. 2007. Mitochondrial point mutations do not limit the natural lifespan of mice. Nat Genet 39:540–543.
Wang X., A.L. Leverin, W. Han, C. Zhu, B.R. Johansson, E. Jacotot, V.S. Ten, et al. 2011. Isolation of brain mitochondria from neonatal mice. J Neurochem 119:1253–1261.
Warner D.A., D.A.W. Miller, A.M. Bronikowski, and F.J. Janzen. 2016. Decades of field data reveal that turtles senesce in the wild. Proc Natl Acad Sci U S A 113:6502–6507.
Wasser S.K.. and D.P.. Barash. 1983. Reproductive Suppression Among Female Mammals : Implications for Biomedicine and Sexual Selection Theory. Q Rev Biol 58:513–538.
Wei Y.H., Y.S. Ma, H.C. Lee, C.F. Lee, and C.Y. Lu. 2001. Mitochondrial theory of aging matures--roles of mtDNA mutation and oxidative stress in human aging. Zhonghua Yi Xue Za Zhi (Taipei).
Whitworth A.J. and L.J. Pallanck. 2009. The PINK1/Parkin pathway: A mitochondrial quality control system? J Bioenerg Biomembr 41:499–503.
Wiley C.D., M.C. Velarde, A.A. Gerencser, E. Verdin, and J. Campisi. 2016. Mitochondrial Dysfunction Induces Senescence with a Distinct Secretory Phenotype. Cell Metab 23:303–314.
Williams G. and B. Charlesworth. 1994. Evolution of Aging Antagonistic Pleiotropy Theory Of Aging (" pay Later " Theory ).
Williams G.C. 1957. Pleiotropy , Natural Selection , and the Evolution of Senescence Author ( s ): George C . Williams. Jstor 11:398–411.
Wright J.H., K.L. Modjeski, J.H. Bielas, B.D. Preston, N. Fausto, L.A. Loeb, and J.S. Campbell. 2011. A random mutation capture assay to detect genomic point mutations in mouse tissue. Nucleic Acids Res 39:1–12.
Wu Z., G. Waneka, A.K. Broz, C.R. King, and D.B. Sloan. 2020. MSH1 is required for maintenance of the low mutation rates in plant mitochondrial and plastid genomes. Proc Natl Acad Sci 117:202001998.
Xu M., T. Pirtskhalava, J.N. Farr, B.M. Weigand, A.K. Palmer, M.M. Weivoda, C.L. Inman, et al. 2018. Senolytics improve physical function and increase lifespan in old age. Nat Med 24:1246–1256.
Yamamoto M. and Y. Takahashi. 2018. The essential role of sirt1 in hypothalamic-pituitary axis. Front Endocrinol (Lausanne) 9:1–6.
Zhang H., L.H. Meng, and Y. Pommier. 2007. Mitochondrial topoisomerases and alternative splicing of the human TOP1mt gene. Biochimie 89:474–481.
Zhang Y., A. Unnikrishnan, S.S. Deepa, Y. Liu, Y. Li, Y. Ikeno, D. Sosnowska, et al. 2017. A new role for oxidative stress in aging: The accelerated aging phenotype in Sod1−/− mice is correlated to increased cellular senescence. Redox Biol 11:30–37.
Ziada A.S., M.Y. Lu, J. Ignas-Menzies, E. Paintsil, M. Li, O. Ogbuagu, S. Saberi, et al. 2019. Mitochondrial DNA somatic mutation burden and heteroplasmy are associated with chronological age, smoking, and HIV infection. Aging Cell 18:1–15.